Circumventing leptin resistance for weight control

Name: Circumventing leptin resistance for weight control
Creator: National Academy of Sciences
Published: 2001-04-10 00:00:00
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PubMed Central2001-04-10 更新2026-05-02 收录

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https://pmc.ncbi.nlm.nih.gov/articles/PMC33319/

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Circumventing leptin resistance for weight control

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National Academy of Sciences

创建时间：

2001-04-10

A Cellular and Molecular Basis of Leptin Resistance

Humans with obesity and diet-induced obese mice (DIO) have high leptin levels and fail to respond to the exogenous hormone suggesting that obesity is caused by leptin resistance, the pathogenesis of w

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Sulforaphane Reduces Obesity by Reversing Leptin Resistance. Sulforaphane Reduces Obesity by Reversing Leptin Resistance

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Leptin-mediated control of body weight by a conserved population of Glp1r-expressing neurons. Leptin-mediated control of body weight by a conserved population of Glp1r-expressing neurons

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

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Body weight, plasma leptin and weight of adipose tissues.

Data are mean ± S.E.M. of 8–10 individual values. MAT (mesenteric adipose tissue), LAT (lumbar adipose tissue), SbAT (Subcutaneous adipose tissue), EpAT (epidimal adipose tissue);* p** p*** pHF ad lib

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A Cellular and Molecular Basis of Leptin Resistance

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