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Dataset S3 from Stromal Senescence By Prolonged CDK4/6 Inhibition Potentiates Tumor Growth

Figshare2017-03-01 更新2026-04-28 收录

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https://figshare.com/articles/dataset/Dataset_S3_from_Stromal_Senescence_By_Prolonged_CDK4_6_Inhibition_Potentiates_Tumor_Growth/22511596 数据链接链接失效反馈

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Cytokine expression in fibroblast conditioned media.

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创建时间：

2017-03-01

肿瘤微环境

基因组学

PurposeTo systematically investigate the characterization of tumor microenvironment (TME) in clear cell renal cell carcinoma (ccRCC), we performed a comprehensive analysis incorporating genomic altera

NIAID Data Ecosystem30

Table1_A decline in avian cytokine expression with age revealed by commercially available cytokine array.XLSX

鸟类免疫

细胞因子表达

Cytokines are secreted immunomodulators that are key regulators of the avian immune response. Currently, the most commonly used method to follow cytokine expression is qPCR, which measures cellular le

NIAID Data Ecosystem00

Basal-to-inflammatory transition enhances basal cell carcinoma (BCC) therapy resistance via crosstalk with a pro-inflammatory stromal niche

基底细胞癌疗法抵抗

肿瘤微环境

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem00

Table_1_Risk Signature of Cancer-Associated Fibroblast–Secreted Cytokines Associates With Clinical Outcomes of Breast Cancer.xlsx

癌症预后预测

肿瘤微环境

Cancer-associated fibroblasts (CAFs) are key components in tumor microenvironment (TME). The secreted products of CAFs play important roles in regulating tumor cells and further impacting clinical pro

NIAID Data Ecosystem30

DataSheet1_Poly I:C-priming of adipose-derived mesenchymal stromal cells promotes a pro-tumorigenic phenotype in an immunocompetent mouse model of prostate cancer.zip

肿瘤微环境

间充质基质细胞

Introduction: Mesenchymal stromal cells (MSC) are envisioned as a potential cellular vehicle for targeted cancer therapies due to their tumor tropism and immune permissiveness. An obstacle in their us

NIAID Data Ecosystem00

Dataset S3 from Stromal Senescence By Prolonged CDK4/6 Inhibition Potentiates Tumor Growth

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