Genetic regulation of FGL2 by rs73375428 and its impact on microglial Aβ clearance in Alzheimer's Disease
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Additional file 7: of Identification of the novel activity-driven interaction between synaptotagmin 1 and presenilin 1 links calcium, synapse, and amyloid beta
Supporting data values. The file presents individual values recorded in the experiments where nâ<â6. (XLSX 270 kb)
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Table_4_Transcriptome Analysis of Pineal Glands in the Mouse Model of Alzheimer’s Disease.XLSX
The pineal gland maintains the circadian rhythm in the body by secreting the hormone melatonin. Alzheimer’s disease (AD) is the most common neurodegenerative disease. Pineal gland impairment in AD is
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Allosteric modulation and structural plasticity of PS2-containing γ-secretase in the production of Aβ and Alzheimer's disease
Alzheimer’s disease (AD) is one of the leading neurodegenerative disorders characterized by amyloid-β (Aβ) plaque accumulation involving the pivotal role of the aspartyl protease γ-secretase. Familial
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Repurposing Triphenylmethane Dyes to Bind to Trimers Derived from Aβ
Soluble oligomers of the β-amyloid peptide, Aβ, are associated with the progression of Alzheimer’s disease. Although many small molecules bind to these assemblies, the details of how these molecules i
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Cell-type specific profiling of human entorhinal cortex at the onset of Alzheimer’s disease neuropathology [RNA-seq]. Cell-type specific profiling of human entorhinal cortex at the onset of Alzheimer’s disease neuropathology [RNA-seq]
Neurons located in the layer II of the entorhinal cortex (ECII) are the primary site of pathological tau accumulation and neurodegeneration at preclinical stages of Alzheimer's disease (AD). Exploring
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