Enhancer Transcripts Mark Active Estrogen Receptor Binding Sites using GRO-seq

ChEBI2013-01-28 更新2026-07-21 收录

激素受体转录组

GRO-seq功能基因组学

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-43836 数据链接链接失效反馈

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资源简介：

Enhancer Transcripts Mark Active Estrogen Receptor Binding Sites using GRO-seq

创建时间：

2013-01-28

Genome-wide transcriptome regulation by progesterone receptor in primary leiomyoma cells

子宫肌瘤分子机制

激素受体转录组

Uterine leiomyomas (LM) affect up to 80% of all reproductive-age women. LM growth requires progesterone, progesterone receptor (PGR), and the maintenance and proliferation of a small (5%) stem cell po

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Genome-wide estrogen receptor-a binding and action in human endometrial stromal cells [RNA-Seq]

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子宫内膜基质细胞分化

Endometrial estrogen receptor-a (ESR1) is indispensable for epithelial and stromal proliferation and differentiation during decidualization, yet the gene targets of estradiol (E2) / ESR1 in human stro

NIAID Data Ecosystem60

Comparison of gene expression profiles for hormone induction in the presence and absence of AP1 binding.

激素受体转录组

染色质可及性分析

Gene expression array analysis component. Ligand-dependent transcription by the nuclear receptor glucocorticoid receptor (GR) is mediated by interactions with co-regulators. The role of these interact

NIAID Data Ecosystem30

A Combination of H2A.Z and H4 Acetylation Recruits Brd2 to Chromatin during Transcriptional Activation

组蛋白变体H2A

激素受体转录组

H2A.Z is an essential histone variant that has been implicated to have multiple chromosomal functions. To understand how H2A.Z participates in such diverse activities, we sought to identify downstream

NIAID Data Ecosystem80

The Androgen Receptor Directly Regulates CD44 Expression in Androgen Sensitive Bladder Cancer [gene expression]

激素受体转录组

膀胱癌基因表达谱

To study androgen receptor transcriptomic activation in the presence of androgen in bladder cancer cell lines. A uniquely derived AR expressing clone of the bladder cancer cell line UMUC3 was derived

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Enhancer Transcripts Mark Active Estrogen Receptor Binding Sites using GRO-seq

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