Molecular Signature of Centrosome Abnormalities in Multiple Myeloma

ChEBI2013-03-21 更新2026-07-21 收录

多发性骨髓瘤病理机制

中心体异常分子标记

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-1038 数据链接链接失效反馈

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资源简介：

Molecular Signature of Centrosome Abnormalities in Multiple Myeloma

创建时间：

2013-03-21

DataSheet_1_Exosomal MiR-4261 mediates calcium overload in RBCs by downregulating the expression of ATP2B4 in multiple myeloma.docx

多发性骨髓瘤病理机制

红细胞研究

BackgroundHypercalcemia induced by multiple myeloma (MM) affects the biological functions of excitable and non-excitable cells. However, red blood cells (RBCs) regulatory effect on calcium in hypercal

NIAID Data Ecosystem50

Additional file 4 of CHEK1 and circCHEK1_246aa evoke chromosomal instability and induce bone lesion formation in multiple myeloma

蛋白质相互作用网络

多发性骨髓瘤病理机制

Additional file 4. Supplementary Table 2 - A list of interacting proteins for CHEK1.

Figshare2021-06-05 更新20

Myeloma formation by RANK signaling and TCL1 in mice highlights implications for human myeloma treatment

多发性骨髓瘤病理机制

RANK信号通路

Multiple myeloma, characterized by the clonal proliferation of malignant plasma cells in the bone marrow, presents significant challenges in treatment, with only a subset of patients achieving long-te

NIAID Data Ecosystem20

Induction of Malignant Plasma Cell Proliferation by Eosinophils

多发性骨髓瘤病理机制

肿瘤微环境

The biology of the malignant plasma cells (PCs) in multiple myeloma (MM) is highly influenced by the bone marrow (BM) microenvironment in which they reside. More specifically, BM stromal cells (SCs) a

NIAID Data Ecosystem40

Induction of Malignant Plasma Cell Proliferation by Eosinophils

多发性骨髓瘤病理机制

微环境细胞互作

Figshare2016-01-18 更新20