Engineered exosomes reprogrammed Gli1+ cells to promote SMC reconstruction and inhibit calcification in TEVBs and autologous pathological vessels. Overall design: After engineered exosome therapy in r

NIAID Data Ecosystem20

Table_1_Crenigacestat (LY3039478) inhibits osteogenic differentiation of human valve interstitial cells from patients with aortic valve calcification in vitro.XLSX

心血管钙化治疗

Notch信号通路

Calcific aortic valve disease (CAVD) is one of the dangerous forms of vascular calcification. CAVD leads to calcification of the aortic valve and disturbance of blood flow. Despite high mortality, the

NIAID Data Ecosystem10

Crenigacestat inhibits osteogenic differentiation of human valve interstitial cells and seems promising for treatment of cardiovascular calcification

心血管钙化治疗

瓣膜间质细胞成骨分化

Vascular calcification is common pathology various forms of which presented in the half of humans. Calcific aortic valve disease (CAVD) is one of the most dangerous forms of vascular calcification. De

NIAID Data Ecosystem40

Details of the use of statins.

药物效果评估

心血管钙化治疗

aNot reported bStatins were given to calcium group at start, to sevelamer group at week 8 only if their LDL-C levels were not less than 70 mg/dL c8% patients were given statins in sevelamer group, whi

NIAID Data Ecosystem20

Comparison of mean BAC and CAC scores among MACE (yes/no).

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