首页/数据集/RNA seq of fluid shear stress treated mouse Pkd1-/- renal epithelial cells

RNA seq of fluid shear stress treated mouse Pkd1-/- renal epithelial cells

ChEBI2019-03-01 更新2026-07-12 收录

多囊肾病机制

转录组力学响应

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-6641 数据链接链接失效反馈

官方服务：

资源简介：

RNA seq of fluid shear stress treated mouse Pkd1-/- renal epithelial cells

应用场景：

创建时间：

2019-03-01

多囊肾病机制

信号通路调控

Cystic kidney disease (CyKD) is the leading monogenetic cause of endstage renal disease. In both mice and humans CyKD has been consistently linked to the activation of the mTORC1 pathway. Yet, the uti

NIAID Data Ecosystem70

mTORC1-mediated growth of kidney cysts is rapidly bypassed by other signaling pathways

多囊肾病机制

信号通路调控

NIAID Data Ecosystem60

Aquaporin-11 knockout effect on kidney. Mus musculus

多囊肾病机制

基因表达谱分析

Aquaporin-11 (AQP11), a new member of the aquaporin family, is localized in the endoplasmic reticulum (ER). Aqp11–/– mice neonatally suffer from polycystic kidneys derived from the proximal tubule. It

NIAID Data Ecosystem30

SE-driven metabolic reprogramming in ADPKD (kidney RNA-seq)

多囊肾病机制

代谢重编程

Metabolic reprogramming is emerging as a key pathological contributor to the progression of autosomal dominant polycystic kidney disease (ADPKD), but the molecular mechanisms underlying dysregulated c

NIAID Data Ecosystem60

Experimental design.

多囊肾病机制

药物靶点发现

Polycystic kidney disease (PKD) is an important cause of kidney failure, but treatment options are limited. While later stages of the disease have been extensively studied, mechanisms driving the init

Figshare2024-12-12 更新40

RNA seq of fluid shear stress treated mouse Pkd1-/- renal epithelial cells

资源简介：

相关数据集