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Colony stimulating factor-1 receptor signaling networks inhibit macrophage inflammatory responses by induction of microRNA-21

ChEBI2014-10-23 更新2026-07-21 收录

巨噬细胞免疫调节

microRNA信号通路

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-62630 数据链接链接失效反馈

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Colony stimulating factor-1 receptor signaling networks inhibit macrophage inflammatory responses by induction of microRNA-21

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创建时间：

2014-10-23

巨噬细胞免疫调节

细胞炎症反应

This experiment explored the impact of cell volume increase and regulatory volume decrease (RVD) on the transcriptome of mouse macrophages. Bone marrow-derived macrophages (BMDMs) were isolated from

NIAID Data Ecosystem80

miRNA array for LPS-activated tnc+/+ and tnc-/- macrophages

巨噬细胞免疫调节

炎症分子机制

Endogenous molecules generated upon pathogen invasion or tissue damage serve as danger signals that activate host defence, however their precise immunological role remains unclear. Tenascin-C is an ex

NIAID Data Ecosystem60

Gene Expression profiling of wild type (WT) and Allograft inflammatory factor-1 deficient (Aif1-/-) immortalized bone marrow derived macrophages by RNA-seq.

转录组学分析

巨噬细胞免疫调节

Purpose: Identification of cellular pathways regulated by AIF1 in bone marrow derived macrophages, based on gene expression patterns identfied by RNA seq. Methods: Imortalized macrophage cell lines we

NIAID Data Ecosystem20

Table 3_TMEM106A mediates atherosclerosis progression through macrophage-centered immune responses and chemokine signaling.xls

动脉粥样硬化机制

巨噬细胞免疫调节

BackgroundAtherosclerosis (AS) remains a leading cause of cardiovascular morbidity and mortality, characterized by intricate interactions between immune dysregulation and lipid metabolism abnormalitie

NIAID Data Ecosystem70

Bmal1 regulates inflammatory response in macrophage

巨噬细胞免疫调节

表观遗传时序分析

Deletion of Arntl disrupted temporal inflammatory response in macrophage. Mechanistically, the acetylation status of lysine 27 of histone 3 (H3K27ac) was enhanced at the PU.1-containing enhancers in A

NIAID Data Ecosystem60

Colony stimulating factor-1 receptor signaling networks inhibit macrophage inflammatory responses by induction of microRNA-21

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