Molecular and Functional Remodeling of the Superior and Inferior Sinoatrial Nodes in a Rat Model of Hypertrophic Cardiomyopathy
收藏Alliance of Genome Resources2026-08-01 收录
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Conclusions: Pressure-overload-induced heart failure resulted in significant downregulation of critical transcription factors, ion channels, and structural transcripts of the iSAN, which could explain the functional silencing of the iSAN in failing rat hearts.
