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Extended-synaptotagmin 1 enhances liver cancer progression mediated by cancer-related unconventional protein secretion

Name: Extended-synaptotagmin 1 enhances liver cancer progression mediated by cancer-related unconventional protein secretion
Creator: Kohji Yamada
Published: 2026-04-09T12:26:01+08:00

Mendeley Data2026-04-09 收录

肝癌分子机制

非经典蛋白质分泌

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https://data.mendeley.com/datasets/djwbyvc9pn 数据链接链接失效反馈

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The unprocessed source data and the statistical source data that support the findings of this study are available.

应用场景：

提供机构：

Kohji Yamada

肝癌分子机制

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Table S5. The significant reduced genes in TET1 knockdown cells. (XLSX 12 kb)

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Understanding the roles of ncRNAs in tumorigenesis and metastasis would establish avenues for the identification of diagnostic and therapeutic targets. We observed that lncRNA SNHG10 and its derived S

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As the third most common cause of cancer-related mortality, hepatocellular carcinoma (HCC) is a global health concern. Despite its prevalence, treatment options are limited, underscoring the need to i

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Development of gene expression signatures in the liver cancer cells with FXR overexpression and FXR activation. Development of gene expression signatures in the liver cancer cells with FXR overexpression and FXR activation

肝癌分子机制

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Astrocyte elevated gene-1 (AEG-1) and c-Myc cooperate to promote hepatocarcinogenesis

肝癌分子机制

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Double transgenic mice with hepatocyte-specific expression of AEG-1 and c-Myc show aggressive HCC compared to single transgenics. Gene expression was analyzed to understand the molecular mechanism by

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Extended-synaptotagmin 1 enhances liver cancer progression mediated by cancer-related unconventional protein secretion

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