RUNX2/CBFB modulates the response to MEK inhibitors through activation of receptor tyrosine kinases in KRAS mutant colorectal cancer
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE139169
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资源简介：
Investigation of the transcriptional consequences of RUNX2 and CBFB loss in the presence and absence of the MEK inhibitor selumetinib in LoVo ERN1 knockout KRAS mutant colorectal cancer cells. RNA-seq comparison of LoVo ERN1 KO, LoVo ERN1/RUNX2 double KO, and LoVo ERN1/CBFB double KO cells treated with 1 uM selumetinib or vehicle.
创建时间：
2020-01-02
