Effect of prolonged exposure to normoxia on miRNA expression of primary mouse chondrocytes

miRNA profiles of primary mouse chondrocytes exposed to prolonged normoxia, which resulted in accumulation of oxidative stress. The sequencing data were generated by miRNA sequencing, in three biological replicates. This study provides insights into the role of chronic oxidative stress in chondrocytes and helps identification of miRNAs related to OA pathogenesis. Overall design: Total RNA was isolated from chondrocytes and subjected to small RNA-seq library preparation using the TruSeq kit (Illumina).