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Transcription profiling of human atherosclerotic lesion SMC cells resistant and sensitive to TGF and Fas ligation by CH11 antibody

ChEBI2008-06-12 更新2026-07-21 收录

动脉粥样硬化机制

平滑肌细胞转录组

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-2522 数据链接链接失效反馈

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Transcription profiling of human atherosclerotic lesion SMC cells resistant and sensitive to TGF and Fas ligation by CH11 antibody

应用场景：

创建时间：

2008-06-12

动脉粥样硬化机制

平滑肌细胞转录组

The multiple claims about reactivation of the embryonic stem cell (ESC) pluripotency factor OCT4 in somatic cells are highly controversial due to the fact that there is no direct evidence that OCT4 ha

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LncRNA ICR regulates endothelium inflammation through feedback activation of NF-κB signaling

动脉粥样硬化机制

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The responsive and abundant lncRNAs to pro-inflammatory factors in human ECs were screened and validated by RNA sequencing and qPCR. ICAM1-related non-coding RNA (ICR) was identified as the most poten

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Comparative transcriptome profiling myometrial and vascular smooth muscle cells. Comparative transcriptome profiling myometrial and vascular smooth muscle cells

平滑肌细胞转录组

靶向药物开发

The goal of this study was to select the optimal myometrial cells for our high-throughput drug discovery assay, as well as determine the similarity or differences of myometrial cells to vascular smoot

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Table_1_Exploration of Crucial Mediators for Carotid Atherosclerosis Pathogenesis Through Integration of Microbiome, Metabolome, and Transcriptome.DOCX

动脉粥样硬化机制

多组学整合分析

BackgroundCarotid atherosclerosis (CAS) is an important cause of stroke. Although interactions between the gut microbiome and metabolome have been widely investigated with respect to the pathogenesis

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Expression data from foam cells of apolipoprotein E-deficient mice. Mus musculus

动脉粥样硬化机制

泡沫细胞

Hypercholesterolemai is a major contributor to atherosclerosis development. To assess the effects of hypercholesterolemia on the transcriptional profiling in foam cells, mice were fed regular chow, or

NIAID Data Ecosystem30

Transcription profiling of human atherosclerotic lesion SMC cells resistant and sensitive to TGF and Fas ligation by CH11 antibody

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