TLR4 Signaling Is a Major Mediator of the Female Tract Response to Seminal Fluid in Mice

ChEBI2015-07-01 更新2026-07-12 收录

生殖免疫学

TLR4信号通路

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TLR4 Signaling Is a Major Mediator of the Female Tract Response to Seminal Fluid in Mice

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2015-07-01

Data_Sheet_1_Immunological Homeostasis at the Ovine Placenta May Reflect the Degree of Maternal Fetal Interaction.docx

生殖免疫学

胎盘免疫调节

Successful mammalian pregnancies are a result of complex physiological, endocrinological, and immunological processes that combine to create an environment where the mother is tolerant to the semi-all

NIAID Data Ecosystem90

Expression data of Human Decidual NK cells, Peripheral blood, CD56Bright NK cells and CD56Dim NK cells on HGU133B arrays. Homo sapiens

NK细胞转录组

生殖免疫学

Fragmented RNA cocktails from FACS sorted Human decidual NK cell, and peripheral blood CD56Bright and CD56Dim NK cells, previously hybridization to HGU95AV2 chips (Koopman et al J Exp Med. 2003 Oct 20

NIAID Data Ecosystem30

Uterine stromal cell_cultured with uNK conditioned Medium

生殖免疫学

子宫内膜细胞互作

The endometrium contains a distinct population of immune cells consisting of 70% natural killer (NK) cells that undergo cyclic changes during the menstrual cycle. However, how these uterine NK (uNK) c

NIAID Data Ecosystem20

Data_Sheet_1_Peptide Tat(48–60) YVEEL protects against necrotizing enterocolitis through inhibition of toll-like receptor 4-mediated signaling in a phosphatidylinositol 3-kinase/AKT dependent manner.pdf

结肠炎治疗

TLR4信号通路

Necrotizing enterocolitis (NEC) is a catastrophic disease largely occurring in preterm infants, and toll-like receptor 4 (TLR4) has been implicated in its pathogenesis. The current therapeutic strateg

NIAID Data Ecosystem80

Table_14_ERAP, KIR, and HLA-C Profile in Recurrent Implantation Failure.docx

生殖免疫学

体外受精

The mother’s uterine immune system is dominated by uterine natural killer (NK) cells during the first trimester of pregnancy. These cells express killer cell immunoglobulin-like receptors (KIRs) of in

NIAID Data Ecosystem20

TLR4 Signaling Is a Major Mediator of the Female Tract Response to Seminal Fluid in Mice

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