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Sensitive mapping of chromatin-altering polymorphisms reveals molecular drivers of autoimmune disease

ChEBI2015-03-01 更新2026-07-21 收录

染色质可及性变异分析

自身免疫病分子机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-58852 数据链接链接失效反馈

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资源简介：

Sensitive mapping of chromatin-altering polymorphisms reveals molecular drivers of autoimmune disease

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创建时间：

2015-03-01

狼疮遗传交互作用

自身免疫病分子机制

Several confirmed genetic susceptibility loci involved in the interferon signaling and Th17/B cell response for SLE in Chinese Han populations have been described. Available data also indicate that se

NIAID Data Ecosystem60

Transcriptome targets of PCBP1 in T cells identified with CLIP. Homo sapiens

细胞转录调控

自身免疫病分子机制

Iron deposition is frequently observed in human autoinflammatory diseases, such as in the brain of patients with multiple sclerosis or in the synovial fluid of patients with rheumatoid arthritis1-5. Y

NIAID Data Ecosystem40

MCC950/CRID3 potently targets the NACHT domain of wild-type NLRP3 but not disease-associated mutants for inflammasome inhibition

炎症小体靶向药物

自身免疫病分子机制

The nucleotide-binding-domain (NBD)–and leucine-rich repeat (LRR)–containing (NLR) family, pyrin-domain–containing 3 (NLRP3) inflammasome drives pathological inflammation in a suite of autoimmune, met

Figshare2019-09-16 更新20

Crystal structure of a MHC class I-peptide complex

抗原呈递结构解析

自身免疫病分子机制

Crystal structure of a MHC class I-peptide complex Descriptor: BETA-2-MICROGLOBULIN, HLA CLASS I HISTOCOMPATIBILITY ANTIGEN, A-3 ALPHA CHAIN, ... Authors: McMahon, R.M, Friis, L, Siebold, C, Friese, M

Protein Data Bank Japan2024-10-16 更新40

Crystal structure of HLA-B*2705 complexed with a peptide derived from the human voltage-dependent calcium channel alpha1 subunit (residues 513-521)

HLA抗原结构解析

自身免疫病分子机制

Crystal structure of HLA-B*2705 complexed with a peptide derived from the human voltage-dependent calcium channel alpha1 subunit (residues 513-521) Descriptor: 9-meric peptide from Voltage-dependent L

Protein Data Bank Japan2024-10-09 更新30

Sensitive mapping of chromatin-altering polymorphisms reveals molecular drivers of autoimmune disease

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