Macrophage-specific inhibition of NF-ÎºB activation impairs foam cell survival

ChEBI2010-06-24 更新2026-07-21 收录

动脉粥样硬化机制

细胞信号通路

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Macrophage-specific inhibition of NF-ÎºB activation impairs foam cell survival

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2010-06-24

The influence of nt-LNA on cell composition and phenotypees in the atherosclerotic lesion.

动脉粥样硬化机制

单细胞RNA测序

Raw fastq files from paired-end, Illumina-based sequencing on NovaSeq6000 Apoe-/- mice were fed a western diet for 4 weeks and administered saline or nt-LNA (30 mg/kg) weekly. After 4 weeks, aortic a

NIAID Data Ecosystem80

Nicotinic Acid Receptor GPR109A Is Down-Regulated in Human Macrophage-Derived Foam Cells

动脉粥样硬化机制

泡沫细胞

Nicotinic acid (NA) regresses atherosclerosis in human imaging studies and reduces atherosclerosis in mice, mediated by myeloid cells, independent of lipoproteins. Since GPR109A is expressed by human

NIAID Data Ecosystem40

The effect of CHRDL2 overexpression on gene expression in CACO2 CRC cells

结肠基因表达

细胞信号通路

Chordin-like-2 (CHRDL2) is a secreted BMP antagonist, with overexpression and genomic variants associated with colorectal cancer (CRC) risk. BMP signalling in the normal intestinal epithelium operates

NIAID Data Ecosystem20

Murine Gammaherpesvirus M2 Protein Induction of IRF4 via the NFAT Pathway Leads to IL-10 Expression in B Cells

疱疹病毒基因调控

细胞信号通路

Reactivation of the gammaherpesviruses Epstein-Barr virus (EBV), Kaposi's sarcoma-associated herpesvirus (KSHV) and murine gammaherpesvirus 68 (MHV68) from latently infected B cells has been linked to

Figshare2016-01-18 更新50

LncRNA ICR regulates endothelium inflammation through feedback activation of NF-κB signaling

动脉粥样硬化机制

血管炎症调控

The responsive and abundant lncRNAs to pro-inflammatory factors in human ECs were screened and validated by RNA sequencing and qPCR. ICAM1-related non-coding RNA (ICR) was identified as the most poten

NIAID Data Ecosystem40

Macrophage-specific inhibition of NF-ÎºB activation impairs foam cell survival

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