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Transcriptome sequencing of spinal cord from tocopherol-transfer protein (alpha) mice

NIAID Data Ecosystem2026-05-17 收录

神经退行性分子机制

营养遗传学转录组学

数据链接：

https://www.ncbi.nlm.nih.gov/sra/SRP120513 数据链接链接失效反馈

官方服务：

资源简介：

Transcriptomic profiles of spinal cord from Ttpa+/+ and Ttpa-/- mice on diets varying in vitamin E concentrations

应用场景：

创建时间：

2017-10-20

衰老相关蛋白

神经退行性分子机制

The attachment of Post-Translational Modifications (PTMs) to proteins plays key roles in the regulation of the activity and stability of various proteins. Here we utilized the nematode Caenorhabditis

NIAID Data Ecosystem40

TDP-43表观遗传学

神经退行性分子机制

This is the summarized excel file containing the original information for all experiment-based figures and supplementary figures in our manuscript "Age-related demethylation of the TDP-43 autoregulato

Figshare2021-01-27 更新10

Expanded gene expression changes in inducible TDP-43 flies.

神经退行性分子机制

TDP-43蛋白相关基因表达

Genes are from same experiment and analysis as in Table 3, with less stringent criteria to identify potentially important genes. nACR, nicotinic acetycholine receptor.

Figshare2015-12-02 更新10

Global gene profiling of (-)-Epigallocatechin-3-gallate (EGCG)-induced neuronal death. Mus musculus

神经退行性分子机制

药物神经保护

Alzheimer’s disease (AD) is the most common form of adult-onset dementia with severe intellectual deterioration and is characterised by the accumulation of the amyloid-β (Aβ) peptides and the presence

NIAID Data Ecosystem30

Additional file 2: Table S2. of Dioxins and related environmental contaminants increase TDP-43 levels

环境毒理学

神经退行性分子机制

(DOCX 28.9 kb)

Figshare2017-05-06 更新10