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Temporal changes of carotid endothelial transcriptome in hyperlipidemic pigs

ChEBI2019-12-31 更新2026-07-12 收录

心血管疾病分子机制

时序转录组分析

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-3776 数据链接链接失效反馈

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资源简介：

Temporal changes of carotid endothelial transcriptome in hyperlipidemic pigs

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创建时间：

2019-12-31

心脏钙火花超分辨率成像

心血管疾病分子机制

This file contains correlative calcium and DNA-PAINT datasets acquired from both Ctrl-RV and MCT-RV groups, custom-written analysis code and output files.

NIAID Data Ecosystem40

Time Series of differentiation from human iAT2s to iAT1s in L+DCI medium

上皮细胞分化

时序转录组分析

The goal of this study is to understand the transcriptomic shift during differentiatiton of iAT2s to iAT1s in 3D BU3 NGAT iPSCs were differentiated into iPSC derived alveolar epithelial type 2 cells (

NIAID Data Ecosystem30

HCM∩CMDF_DMNC_top6

心血管疾病分子机制

网络相互作用分析

Top 6 elements of the intersection of molecular interactions of hypertrophic cardiomyopathy and coronary microvascular dysfunction ranked by DMNC centrality

NIAID Data Ecosystem20

Supplementary Material for: Calreticulin Regulates Neointima Formation and Collagen Deposition following Carotid Artery Ligation

血管平滑肌细胞生物学

心血管疾病分子机制

Background/Aims: The endoplasmic reticulum (ER) stress protein, calreticulin (CRT), is required for the production of TGF-β-stimulated extracellular matrix (ECM) by fibroblasts. Since TGF-β regulates

Figshare2017-06-20 更新30

Raw sequence reads

心血管疾病分子机制

转录组学分析

To investigate the potential pathogenic role of SLC25A3 defects in myocardial hypertrophy, we performed RNA sequencing on WT and KO cell groups after culturing hiPSC-CMs.

NIAID Data Ecosystem20

Temporal changes of carotid endothelial transcriptome in hyperlipidemic pigs

资源简介：

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