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HIV-1 gp120 impairs B cell proliferation by inducing TGF-β1 production and FcRL4 expression via an α4β7-dependent mechanism

ChEBI2013-10-17 更新2026-07-21 收录

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B细胞功能抑制

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-49019 数据链接链接失效反馈

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HIV-1 gp120 impairs B cell proliferation by inducing TGF-β1 production and FcRL4 expression via an α4β7-dependent mechanism

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创建时间：

2013-10-17

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决策树模型

Figure S1 and Table S1. Figure S1, “plotcp” function allows to plot mean and standard deviation of the errors in the cross-validated prediction of the “rpart” object shown in Figure 5 of the main docu

Figshare2015-12-02 更新110

HIV specific CD8+ Trm-like cells in tonsils express exhaustive signatures in the absence of natural HIV control

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组织驻留记忆T细胞

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem70

LDGs and NDGs in HIV+ patients in CD4high HIV+ patients.

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流式细胞表型分析

Ldgs And Ndgs Were Isolated From The Blood Of Hiv+ Patients With Cd4+ T Cell Counts >350 Cells/µL (N = 11) As Described In Materials And Methods. Expression Levels Of Phenotypic Markers Were Determine

NIAID Data Ecosystem60

ATN 026 Antiretroviral Medications - Other Antiretroviral Medications Details [CRF 8] Dataset in Evaluation of HIV-Specific CD8+ T-Cell Responses and Escape Mutations as Explanations for the Observed Differences in Disease Progression Conferred by HLA Class I Alleles

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HLA与病毒性疾病关联

Data from Antiretroviral Medications Form - Other antiretroviral medications details [CRF 8] Study Description ATN 026 investigated the association between HLA class I genotypes and differing HIV-1 di

NIAID Data Ecosystem110

MOESM1 of Lymphocyte homeostasis is maintained in perinatally HIV-infected patients after three decades of life

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细胞稳态

Additional file 1. Gender-related median values of TRECs and KRECs in perinatally HIV-infected youths (pHIVy), in non-perinatally HIV-infected youths (npHIVy) and in healthy controls (HC).

NIAID Data Ecosystem60

HIV-1 gp120 impairs B cell proliferation by inducing TGF-β1 production and FcRL4 expression via an α4β7-dependent mechanism

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