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资源简介:
ChIP-seq INA-6
应用场景:
创建时间:
2025-12-29
相关数据集
BET inhibition releases the Mediator complex from specific cis elements in acute myeloid leukemia cells (ChIP-Seq II)
Genome occupancy profiling by high throughput sequencing ChIP-seq of Brd4 with or without Doxycycline treatment to induce Med12 or Med23 shRNAs in MLL-AF9 transformed acute myeloid leukemia cells (RN2
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Genome-wide maps of chromatin state in PANC-1 cells.
To investigate functional associations between SIRT1 and CUL4B, we used ChIP-seq to analyze genome-wide SIRT1/CUL4B complex transcriptional targets. ChIP experiments were performed first in PANC-1 ce
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Mapping ERβ genomic binding sites reveals unique genomic features and identifies EBF1 as an ERβ interactor [Gro-Seq]. Homo sapiens
The C4-12/Flag.ERβ cell line which stably expressed Flag.ERβ is used to study ERβ genomic functions without ERα interference. Mapping ERβ binding sites in these cells reveals ERβ unique distribution a
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Investigation of Rap1 binding occupancy in global H3-depletion condition [Rap1_ChIPseq]. Investigation of Rap1 binding occupancy in global H3-depletion condition [Rap1_ChIPseq]
We constructed the inducible H3-depleted and FLAG-tag of the repressor activator protein 1 (Rap1) yeasts to investigate the depleted effect of nucleosomal H3 on DNA-binding profiles of Rap1 and result
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