Effects of LMTK2 knockdown on total RNA expression in CFBE41o- cells after TGFβ1 or vehicle control (HCl+BSA) treatment

The purpose of this study was to increase the current knowledge about the LMTK2 network and its intersection with the TGF-β1 signaling pathway. To understand how the LMTK2 and TGF-β1 pathways interconnect, we knocked down LMTK2 using small(si)RNA-mediated silencing in human bronchial epithelial CFBE41o- cells, treated cells with TGF-β1 or vehicle control, and performed differential gene expression analysis by RNA sequencing (RNAseq). Differential gene expression analysis by RNA sequencing (RNAseq) in parental CFBE41o- cells after LMTK2 knockdown and 24hour treatment with TGFβ1 or vehicle control (HCl+BSA)