Cdk1 activity levels drive periodic transcription independently of cell cycle progression

ChEBI2016-04-01 更新2026-07-12 收录

细胞周期调控

周期性转录机制

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-70717 数据链接链接失效反馈

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Cdk1 activity levels drive periodic transcription independently of cell cycle progression

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2016-04-01

Additional file 16 of STAU2 protein level is controlled by caspases and the CHK1 pathway and regulates cell cycle progression in the non-transformed hTERT-RPE1 cells

细胞周期调控

蛋白相互作用分析

Additional file 16: Table S9. Gene ontology (GO) of STAU2 interactors that show reduced proximity to STAU2 in hTERT-RPE1 cells treated with the CHK1 inhibitor PF47 compared to untreated cells. List of

Figshare2021-03-05 更新60

Arrest of G(1)-S Progression by the p53-Inducible Gene PC3 Is Rb Dependent and Relies on the Inhibition of Cyclin D1 Transcription

细胞周期调控

肿瘤抑制基因

The p53-inducible gene PC3 (TIS21, BTG2) is endowed with antiproliferative activity. Here we report that expression of PC3 in cycling cells induced accumulation of hypophosphorylated, growth-inhibitor

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Cumulative Effect of Phosphorylation of pRB on Regulation of E2F Activity

细胞周期调控

磷酸化机制

The product of the retinoblastoma susceptibility gene, pRB, is a nuclear phosphoprotein that controls cell growth by binding to and suppressing the activities of transcription factors such as the E2F

PubMed Central20

A kinase-phosphatase switch transduces environmental information into a bacterial cell cycle circuit. Caulobacter vibrioides

细胞周期调控

环境胁迫信号转导

The bacterial cell cycle has been extensively studied under standard growth conditions. How it is modulated in response to environmental changes remains poorly understood. Here, we demonstrate that th

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DataSheet4_PRR11 promotes cell proliferation by regulating PTTG1 through interacting with E2F1 transcription factor in pan-cancer.zip

癌症分子机制

细胞周期调控

The upregulated proline rich 11 (PRR11) plays a critical role in cancer progression. The relevant biological functions of PRR11 in pan-cancer development are not well understood. In the current study,

NIAID Data Ecosystem30

Cdk1 activity levels drive periodic transcription independently of cell cycle progression

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