Acinetobacter baumannii gene expression in the presence of calprotectin

Calprotectin (CP) inhibits bacterial viability through extracellular chelation of transition metals. However, how CP influences general metabolism remains largely unexplored. We show here that CP restricts bioavailable Zn and Fe to the pathogen Acinetobacter baumannii, inducing an extensive multi-metal perturbation of cellular physiology. Detailed here are the RNA sequencing files of WT A. baumannii ATCC 17978 grown plus or minus recombinant human calprotectin. Overall design: Growth of three biological replicates of A. baumannii ATCC 17978 plus or minus 250 microgams/ml CP in lysogeny broth (LB) supplemented with CP buffer.