Cooperation between Androgen Receptor and Polycomb in oncogenic transformation

ChEBI2011-12-11 更新2026-07-21 收录

癌症分子机制

表观遗传调控

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-31978 数据链接链接失效反馈

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Cooperation between Androgen Receptor and Polycomb in oncogenic transformation

创建时间：

2011-12-11

DataSheet4_PRR11 promotes cell proliferation by regulating PTTG1 through interacting with E2F1 transcription factor in pan-cancer.zip

癌症分子机制

细胞周期调控

The upregulated proline rich 11 (PRR11) plays a critical role in cancer progression. The relevant biological functions of PRR11 in pan-cancer development are not well understood. In the current study,

NIAID Data Ecosystem30

DNA dioxygenases Tet2/3 control epithelial differentiation [RNA-seq]

毛囊上皮分化

表观遗传调控

Execution of lineage-specific differentiation programs requires tight coordination between many regulators including Ten-eleven translocation (TET) family enzymes catalyzing 5-methylcytosine oxidation

Alliance of Genome Resources00

DNMT3A Reads and Connects Histone H3K36me2 to DNA methylation [RNA-seq]

表观遗传调控

甲基化反应机制

DNA methylation at the 5-position of cytosine (5mC) is a crucial epigenetic mark in regulating biological processes including gene silencing, gene imprinting, and X chromosome inactivation. It has lon

NIAID Data Ecosystem00

Table_1_ABCC6 Knockdown Fuels Cell Proliferation by Regulating PPARα in Hepatocellular Carcinoma.xls

癌症分子机制

ABCC6基因功能

The ATP binding cassette (ABC) transporter family is ubiquitous in eukaryotes, specifically in vertebrates, and plays a crucial role in energy homeostasis, cell signaling, and drug resistance. Accumul

NIAID Data Ecosystem20

Heterogeneity study on miRNAs expression in islet cells of rat pancreatic head and tail

胰腺头尾miRNA异质性

癌症分子机制

Background:As a vital endocrine organ, the pancreas, which has differences in terms of anatomical structure and microenvironment between the pancreatic head and tail. However, it is currently unclear

NIAID Data Ecosystem00

Cooperation between Androgen Receptor and Polycomb in oncogenic transformation

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