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Changes in global gene expression profiles induced by HPV 16 E6 oncoprotein variants in cervical carcinoma C33-A cells

ChEBI2015-11-20 更新2026-07-12 收录

HPV致癌机制

宫颈癌基因表达

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-73761 数据链接链接失效反馈

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Changes in global gene expression profiles induced by HPV 16 E6 oncoprotein variants in cervical carcinoma C33-A cells

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创建时间：

2015-11-20

甲基化测序

HPV致癌机制

Presented in comparison between groups of differing age, screening outcome, and HPV status. The HPV groups presented were positive vs. negative genotyping test, single vs. multiple genotypes within po

Figshare2022-09-22 更新70

Long non-coding RNA FAM83H-AS1 is regulated by human papillomavirus 16 E6 independently of p53 in cervical cancer cells

HPV致癌机制

lncRNA

Global long non-coding RNA (lncRNA) expression profile in primary human keratinocytes post-expression of high-risk HPV-16 E6 oncoprotein Purpose: The goal of this study is to utlize high-throughput RN

NIAID Data Ecosystem30

Identification of deregulated pathways, key regulators, and novel miRNA-mRNA interactions in HPV-mediated transformation. [CGH cell lines-Agilent]. Identification of deregulated pathways, key regulators, and novel miRNA-mRNA interactions in HPV-mediated transformation. [CGH cell lines-Agilent]

HPV致癌机制

分子生物学

Next to a persistent infection with high-risk human papillomavirus (HPV), molecular changes are required for the development of cervical cancer. To identify which molecular alterations drive carcinoge

NIAID Data Ecosystem20

Human HPV16 positive CaSki cells: Control vs. virus oncogene e6/e7 silenced

HPV致癌机制

肿瘤基因功能验证

Human HPV16 positive CaSki cells: Control vs. virus oncogene e6/e7 silenced

ChEBI2016-07-01 更新40

Effect of HPV E5 on tumor on immune response and tumor development

肿瘤免疫微环境

HPV致癌机制

To investigate the functions of HPV16 E5, we established empty vector or HPV16 E5-transduced CAL-27 and DC2.4 cell lines. We then performed gene expression profiling analysis using data obtained from

NIAID Data Ecosystem30

Changes in global gene expression profiles induced by HPV 16 E6 oncoprotein variants in cervical carcinoma C33-A cells

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