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Transcription profiling by array of wound samples from rats treated with the 5alpha-reductase inhibitor MK-434

ChEBI2009-02-20 更新2026-07-21 收录

伤口愈合分子机制

药物干预转录组

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MEXP-1232 数据链接链接失效反馈

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资源简介：

Transcription profiling by array of wound samples from rats treated with the 5alpha-reductase inhibitor MK-434

应用场景：

创建时间：

2009-02-20

代谢紊乱机制

药物干预转录组

Purpose: a transcriptome analysis was used to ascertain whether lipid metabolism disorders were induced by high-fat diet (HFD), and comparisons were made between control, HFD and DfCS-Ib(treatment) gr

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Mus musculus Transcriptome or Gene expression

药物干预转录组

组织基因表达调控

Transcriptome sequencing results of epididymal adipose tissue from mice on a high-fat diet supplemented with nuciferine and mice on a high-fat diet without nuciferine. To investigate the effect of nuc

NIAID Data Ecosystem40

Supplementary Material for: The Drosophila Chitinase-Like Protein IDGF3 Is Involved in Protection against Nematodes and in Wound Healing

昆虫先天免疫

伤口愈合分子机制

Chitinase-like proteins (CLPs) of the 18 glycosyl hydrolase family retain structural similarity to chitinases but lack enzymatic activity. Although CLPs are upregulated in several human disorders that

Figshare2017-06-20 更新30

Gene expression profiling of myofibroblasts in young and aged day 5 wound beds. Gene expression profiling of myofibroblasts in young and aged day 5 wound beds

伤口愈合分子机制

成纤维细胞亚群

Mesenchymal cells were isolated from day 5 dorsal skin wound beds of 7-weeks old and >24-months old using FACS. This experiment describes multiple unique subsets of wound bed myofibrolbasts capable of

NIAID Data Ecosystem80

Identification the proteins interacted with SNHG26 in human keratinocytes

蛋白质相互作用鉴定

伤口愈合分子机制

The cell transition from an inflammatory phase to a subsequent proliferative phase is crucial for wound healing, yet the driving mechanism remains unclear. By profiling lncRNA expression changes durin

NIAID Data Ecosystem30

Transcription profiling by array of wound samples from rats treated with the 5alpha-reductase inhibitor MK-434

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