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The absence of SEMA7A enhances antitumor immunity against breast cancer by suppressing KDM4A-mediated DNA replication stress and activating CD8+ T cells

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NIAID Data Ecosystem2026-05-10 收录
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https://www.ncbi.nlm.nih.gov/sra/SRP644666
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资源简介:
This study explored the molecular mechanism of SEMA7A affecting DNA replication stress and further regulating the recruitment and activation of CD8+T cells by targeting KDM4A, thus affecting the anti-tumor immunity of breast cancer
创建时间:
2025-11-16
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