首页/数据集/CaM Kinase II mediates maladaptive post-infarct remodeling but not acute myocardial ischemia/reperfusion injury

CaM Kinase II mediates maladaptive post-infarct remodeling but not acute myocardial ischemia/reperfusion injury

ChEBI2014-09-08 更新2026-07-21 收录

缺血再灌注损伤机制

心血管分子病理学

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-58486 数据链接链接失效反馈

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CaM Kinase II mediates maladaptive post-infarct remodeling but not acute myocardial ischemia/reperfusion injury

应用场景：

创建时间：

2014-09-08

缺血再灌注损伤机制

组学靶点发现

Myocardial I/R injury models were established using SPF-grade male Sprague-Dawley (SD) rats, randomly divided into control, I/R, and I/R+Nicorandil groups (n=6 per group). The I/R+Nicorandil group, ni

NIAID Data Ecosystem60

DataSheet1_Anti-Myocardial Ischemia Reperfusion Injury Mechanism of Dried Ginger-Aconite Decoction Based on Network Pharmacology.zip

中药网络药理学

缺血再灌注损伤机制

Dried ginger-aconite decoction (DAD) is a traditional Chinese medicine (TCM) formula that has been extensively used in the treatment of myocardial ischemia reperfusion injury (MI/RI). However, its spe

NIAID Data Ecosystem40

Additional file 3 of Comprehensive analysis of the cardiac proteome in a rat model of myocardial ischemia-reperfusion using a TMT-based quantitative proteomic strategy

心脏蛋白质组学

缺血再灌注损伤机制

Additional file 3. PANTHER analysis results of the differentially expressed proteins in IR injury.

Figshare2020-03-07 更新20

Table9_Activation of CCL21-GPR174/CCR7 on cardiac fibroblasts underlies myocardial ischemia/reperfusion injury.xlsx

缺血再灌注损伤机制

心脏保护靶点发现

Background: The mechanisms underlying myocardial ischemia/reperfusion (I/R) injury are not fully understood. This study aims to explore key candidate genes and potential therapeutic targets for treatm

NIAID Data Ecosystem40

Supplementary Material for: Moderate Hydrogen Peroxide Postconditioning Ameliorates Ischemia/Reperfusion Injury in Cardiomyocytes via STAT3-Induced Calcium, ROS, and ATP Homeostasis

缺血再灌注损伤机制

STAT3信号通路

Introduction: Moderate hydrogen peroxide postconditioning (H2O2PoC) activates signal transducer and activator of transcription 3 (STAT3) to alleviate mitochondrial calcium overload during cardiac isch

NIAID Data Ecosystem30

CaM Kinase II mediates maladaptive post-infarct remodeling but not acute myocardial ischemia/reperfusion injury

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