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gp130/STAT3 signaling is required for homeostatic proliferation and anabolism in postnatal growth plate and articular chondrocytes

NIAID Data Ecosystem2026-03-13 收录

软骨细胞稳态调控

STAT3信号通路

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE188354 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2022-02-09

肿瘤药理学

STAT3信号通路

Colitis-associated colorectal cancer (CAC) is a specific type of colorectal cancer (CRC) with high mortality and morbidity, the chronic inflammation in the intestinal mucosal is the characteristic of

NIAID Data Ecosystem40

Table_1_Combination of Auranofin and ICG-001 Suppress the Proliferation and Metastasis of Colon Cancer.docx

结肠靶向

STAT3信号通路

Colon cancer is one of the deadliest tumors in the world, and with high metastasis rate and mortality, effective drugs for its treatment are still in need. Auranofin (AF) is a gold complex that has be

NIAID Data Ecosystem30

CDYL2 epigenetically regulates MIR124 to control NF-kB/STAT3-dependent breast cancer cell plasticity [RNA-seq]

乳腺癌细胞可塑性

STAT3信号通路

Epigenetic deregulation of gene transcription is central to cancer cell plasticity and malignant progression, but remains poorly understood. We found that the uncharacterized epigenetic factor Chromod

NIAID Data Ecosystem30

Table1_Genetic deficiency of protein inhibitor of activated STAT3 suppresses experimental abdominal aortic aneurysms.docx

主动脉瘤病理机制

STAT3信号通路

AimSignal transducer and activator of transcription (STAT) signaling is critical for the pathogenesis of abdominal aortic aneurysms (AAAs). Though protein inhibitor of activated STAT3 (PIAS3) negative

NIAID Data Ecosystem40

Supplementary Information II.pdf

STAT3信号通路

蛋白质翻译后修饰

Supplementary information for the paper: Asymmetric post-translational modifications regulate the intracellular distribution of unstimulated STAT3 dimers Beatriz Cardoso, Ricardo Letra-Vilela,

Figshare2019-02-22 更新30

gp130/STAT3 signaling is required for homeostatic proliferation and anabolism in postnatal growth plate and articular chondrocytes

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