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MYC couples SWI/SNF and metabolic reprogramming to sustain minimal residual disease in mTORC1-activated cancer cells

NIAID Data Ecosystem2026-05-02 收录

肿瘤代谢重组

mTORC1信号通路

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE270779 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2025-06-10

mTORC1信号通路

RagGTPase调控

CryoEM structure of the SLC38A9-RagA-RagC-Ragulator complex in the post-GAP state Descriptor: GUANOSINE-5'-DIPHOSPHATE, Ragulator complex protein LAMTOR1, Ragulator complex protein LAMTOR2, ... Author

Protein Data Bank Japan2024-03-06 更新80

Mutant SRSF2-associated impaired erythropoiesis is defined by increased mTORC1 signaling due to FYN missplicing

红细胞生成障碍

mTORC1信号通路

Using mouse models and primary human samples, we investigated the impact of SRSF2 mutations on erythropoiesis. We found reduced erythropoiesis in Srsf2P95H versus wild-type mice upon stress-induced er

NIAID Data Ecosystem50

Gene expression profiling in RagC T89N germinal center B cells and RagC S74C murine B220+ lymphoma cells

滤泡性淋巴瘤分子机制

mTORC1信号通路

Gene expression by RNAseq in murine germinal centers induced by SRBC in RagC T89N mice (YRT codes) and RNAseq in murine B220+ from VavVPBcl2/RagC S74C and VavVPBcl2/RagC +/+ lymphomas Follicular Lymp

NIAID Data Ecosystem50

KAT7-mediated CANX (calnexin) crotonylation regulates leucine-stimulated MTORC1 activity

mTORC1信号通路

蛋白质修饰

Amino acids play crucial roles in the MTOR (mechanistic target of rapamycin kinase) complex 1 (MTORC1) pathway. However, the underlying mechanisms are not fully understood. Here, we establish a cell-f

DataCite Commons2025-01-27 更新80

Gene expression data from control and REPTOR (=CG13624) KO male adults

基因表达调控

mTORC1信号通路

The aim was to identify genes whose transcription is induced or repressed by REPTOR (=CG13624) KO in Drosophila melanogaster male adults. Data are part of the manuscript REPTOR and REPTOR-BP regulate

NIAID Data Ecosystem50

MYC couples SWI/SNF and metabolic reprogramming to sustain minimal residual disease in mTORC1-activated cancer cells

资源简介：

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