Evidence of epigenetic reprogramming in bone marrow following influenza A (H1N1) infection during early life

ChEBI2025-12-31 更新2026-07-12 收录

感染免疫表观遗传学

H1N1流感早期感染模型

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-14387 数据链接链接失效反馈

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Evidence of epigenetic reprogramming in bone marrow following influenza A (H1N1) infection during early life

创建时间：

2025-12-31

Intergenerational inheritance of trained immunity and heterologous resistance to infections (RRBS - DNA methylation). Intergenerational inheritance of trained immunity and heterologous resistance to infections (RRBS - DNA methylation)

训练免疫力跨代遗传

感染免疫表观遗传学

Intergenerational inheritance (Inter-I) of phenotypic characteristics has been demonstrated in plants and invertebrates, with epigenetic modifications playing a role in the inheritance of a variety of

NIAID Data Ecosystem90

Gene activation precedes DNA demethylation in response to infection in human dendritic cells

感染免疫表观遗传学

树突状细胞免疫应答

DNA methylation is considered to be a relatively stable epigenetic mark. Yet, a growing body of evidence indicates that DNA methylation levels can change rapidly, for example, in innate immune cells f

NIAID Data Ecosystem50

Durable Alterations of Hematopoiesis and Chromatin post-COVID-19 [MultiomeATAC-seq]. Durable Alterations of Hematopoiesis and Chromatin post-COVID-19 [MultiomeATAC-seq]

感染免疫表观遗传学

干细胞研究

Acute SARS-CoV-2 infection is often highly inflammatory and protracted. Recent advances have established that inflammation can trigger innate immune memory and a persistent influence on hematopoietic

NIAID Data Ecosystem40

Alteration of gene expression in mouse mammary tissue upon E. coli infection. Mus musculus

牛乳腺炎致病机制

感染免疫表观遗传学

Mastitis is a multietiological complex disease, which is defined as inflammation of parenchyma of mammary glands. Bacterial infection is the predominant cause of mastitis, though fungal, viral and myc

NIAID Data Ecosystem60

Enhancer-instructed chromatin folding and compartmentalization dictate Vκ repertoire rearrangement to combat bacterial infection [Hi-C]. Enhancer-instructed chromatin folding and compartmentalization dictate Vκ repertoire rearrangement to combat bacterial infection [Hi-C]

染色质构象与抗体基因重排

感染免疫表观遗传学

Antigen receptor loci are organized into variable (V), diversity (D), and joining (J) elements that rearrange to generate diverse antigen receptor repertoires. Here, we identified an enhancer (E34) in

NIAID Data Ecosystem40

Evidence of epigenetic reprogramming in bone marrow following influenza A (H1N1) infection during early life

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