VHL Status Influences Phenotype of Liver Cancers Arising from PTEN Loss
收藏官方服务:
资源简介:
VHL Status Influences Phenotype of Liver Cancers Arising from PTEN Loss
应用场景:
创建时间:
2014-10-31
相关数据集
Additional file 7: of Circular RNA circTRIM33â12 acts as the sponge of MicroRNA-191 to suppress hepatocellular carcinoma progression
Table S5. The significant reduced genes in TET1 knockdown cells. (XLSX 12 kb)
NIAID Data Ecosystem130
Gene expression profiling of HCCLM3 cells: Control ASO vs SCARNA13 ASO
Understanding the roles of ncRNAs in tumorigenesis and metastasis would establish avenues for the identification of diagnostic and therapeutic targets. We observed that lncRNA SNHG10 and its derived S
NIAID Data Ecosystem30
CRTC2 Forms Co-condensates with YTHDF2 that Enhance Translational Efficiency of m6A-Modified mRNAs to Drive Hepatocarcinogenesis and Lenvatinib Resistance (RNA-seq)
As the third most common cause of cancer-related mortality, hepatocellular carcinoma (HCC) is a global health concern. Despite its prevalence, treatment options are limited, underscoring the need to i
NIAID Data Ecosystem20
Development of gene expression signatures in the liver cancer cells with FXR overexpression and FXR activation. Development of gene expression signatures in the liver cancer cells with FXR overexpression and FXR activation
To investigate the role of nuclear receptor FXR during hepatocarcinogenesis, HepG2 cels and SK-Hep-1 cells were transfected with lentiviral mediated FXR overexpressive vector or the negative control.
NIAID Data Ecosystem50
Astrocyte elevated gene-1 (AEG-1) and c-Myc cooperate to promote hepatocarcinogenesis
Double transgenic mice with hepatocyte-specific expression of AEG-1 and c-Myc show aggressive HCC compared to single transgenics. Gene expression was analyzed to understand the molecular mechanism by
NIAID Data Ecosystem30



