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资源简介：
Transcription profiling by array of kinin-dependent genes in endometrial cancer
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创建时间：
2025-01-24
相关数据集
Chromatin Immunoprecipitation(ChIP)-seq of TCF19 in endometrial cancer cells
In our study, TCF19 was found to be upregulated in microsatellite instability (MSI) endometrial cancer and was associated with poor prognosis and immune exhaustion signature. Elevated TCF19 enhanced
NIAID Data Ecosystem50
ARID1A and PI3-Kinase pathway mutations in the endometrium drive epithelial transdifferentiation and collective invasion [12Z_1A_PI3K_RNA-seq]. ARID1A and PI3-Kinase pathway mutations in the endometrium drive epithelial transdifferentiation and collective invasion [12Z_1A_PI3K_RNA-seq]
ARID1A and PI3-Kinase (PI3K) pathway alterations are common in neoplasms originating from the uterine endometrium. Here we show that monoallelic loss of ARID1A in the mouse endometrial epithelium is s
NIAID Data Ecosystem40
Clinical and genomic crosstalk between glucocorticoid receptor and estrogen receptor a in endometrial cancer [ChIP-seq]
Steroid hormone receptors are simultaneously active in many tissues and capable of altering each other''s function. Estrogen receptor ? (ER) and glucocorticoid receptor (GR) are expressed in the uteru
NIAID Data Ecosystem20
ARID1A and PI3-Kinase pathway mutations in the endometrium drive epithelial transdifferentiation and collective invasion [12Z_ATAC-seq]
ARID1A and PI3-Kinase (PI3K) pathway alterations are common in neoplasms originating from the uterine endometrium. Here we show that monoallelic loss of ARID1A in the mouse endometrial epithelium is s
NIAID Data Ecosystem40
Breeding trial summary.
Chromatin remodeling plays an integral part in endometrial homeostasis through its roles in the maintenance of cell identity, epithelial integrity, and prevention of endometrial disease. Chromodomain-
NIAID Data Ecosystem30
