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Additional file 2: of Bioinformatics analysis of glial inflammatory responses to air pollution

Name: Additional file 2: of Bioinformatics analysis of glial inflammatory responses to air pollution
Creator: Jiang, Wei; Tang, Nina; Xu, Yan; Li, Chenyu
Published: 2019-10-24T00:00:00+08:00

Figshare2024-02-08 更新2026-04-08 收录

神经胶质炎症反应

空气污染毒理学

数据链接：

https://springernature.figshare.com/articles/dataset/Additional_file_2_of_Bioinformatics_analysis_of_glial_inflammatory_responses_to_air_pollution/10035302/1 数据链接链接失效反馈

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资源简介：

Differentially expressed RNAs between â nPM-treatedâ and control groups. (CSV 167 kb)

应用场景：

提供机构：

Jiang, Wei; Tang, Nina; Xu, Yan; Li, Chenyu

创建时间：

2019-10-24

PM2.5暴露翻译调控

空气污染毒理学

We report the application of Ribosome sequencing for high-throughput profiling of tanslation efficiency in BEAS-2B cells after exposure to PM2.5. Overall design: Comparison of tanslation efficiency be

NIAID Data Ecosystem60

8-oxoG RNA IP data set 2: Air pollution mixture derived from the reaction of 100 ppb acrolein, 97 ppb methacrolein, 44 ppb alpha-pinene and 100 ppb O3 for 1.5 h. 8-oxoG RNA IP data set 2: Air pollution mixture derived from the reaction of 100 ppb acrolein, 97 ppb methacrolein, 44 ppb alpha-pinene and 100 ppb O3 for 1.5 h

RNA氧化

空气污染毒理学

While environmental agents such as air pollution have been shown to be causative to diseases, yet limited knowledge exist on their mechanism of action. Here we used RNA-sequencing in combination with

NIAID Data Ecosystem50

Additional file 11: of Transcriptional profiling of human bronchial epithelial cell BEAS-2B exposed to diesel and biomass ultrafine particles

空气污染毒理学

转录组学

Table S7. Genes identified by maSigPro as differentially modulated in time during the exposure to biomass or diesel UFP, respectively. (XLS 66Â kb)

NIAID Data Ecosystem30

In vitro Evaluation of Oxidative Stress Caused by Fine Particles (PM2.5) Exhausted from Heavy-Duty Vehicles Using Diesel/Biodiesel Blends under Real World Conditions

空气污染毒理学

生物柴油排放评估

In this work, the redox activity of fine diesel/biodiesel particulate matter (PM2.5) was studied in order to approach its toxicity from reactive oxygen species, due to adverse effects it may cause to

NIAID Data Ecosystem60

Transcriptomic analysis of endothelial progenitor cells (EPCs) isolated from mice exposed to concentrated air pollution particles (CAPS) [mRNA-seq]. Transcriptomic analysis of endothelial progenitor cells (EPCs) isolated from mice exposed to concentrated air pollution particles (CAPS) [mRNA-seq]

转录组学分析

空气污染毒理学

Mice were exposed to filtered air or CAPs for 30 days and then bone marrow-derived EPCs isolated and cultured for 12 days. RNAs isolated from these mice were used for RNA seq analysis. Overall design:

NIAID Data Ecosystem30

Additional file 2: of Bioinformatics analysis of glial inflammatory responses to air pollution

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