EWS-FLI1 and HOXD13 control tumor cell plasticity in Ewing sarcoma

NIAID Data Ecosystem2026-03-13 收录

尤文肉瘤分子机制

癌细胞可塑性

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE182513 数据链接链接失效反馈

官方服务：

资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

创建时间：

2022-07-01

CD99 inhibits neural differentiation of human Ewing sarcoma cells

尤文肉瘤分子机制

神经分化调控

Ewing sarcoma (EWS) is an aggressive bone tumor of uncertain cellular origin. CD99 is a membrane protein that is expressed in most cases of EWS, although its function in the disease is unknown. Here w

NIAID Data Ecosystem110

Supplementary Table S6 from KDM6 Demethylases Contribute to EWSR1::FLI1-Driven Oncogenic Reprogramming in Ewing Sarcoma

尤文肉瘤分子机制

表观遗传调控

Excel file showing DEG and downregulated direct targets upon KDM6A or KDM6B knockout.

Figshare2025-11-14 更新60

EWS-FLI1 represses Rho-actin signaling via MRTFB/YAP-1/TEAD perturbation in Ewing Sarcoma [Chip-Seq]. Homo sapiens

尤文肉瘤分子机制

转录调控网络

Ewing Sarcoma (EwS) is a EWS-FLI1- fusion driven pediatric bone cancer with high metastatic potential. Cellular plasticity, typically regulated via the Rho-pathway, is a prerequisite for metastasis in

NIAID Data Ecosystem40

Reciprocal inhibition of NOTCH and SOX2 shapes tumor cell plasticity and therapeutic escape in triple negative breast cancer.

三阴性乳腺癌耐药性

癌细胞可塑性

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem70

Paracrine Ras/ERK signalling drives gene expression plasticity in a model of basal-like breast cancer. Paracrine Ras/ERK signalling drives gene expression plasticity in a model of basal-like breast cancer

癌细胞可塑性

单细胞信号动力学

The EGFR/Ras/ERK signalling pathway is a primary driver of cancer cell proliferation and metastasis in tumours that exhibit high cell-to-cell heterogeneity. While the signalling activity of this pathw

NIAID Data Ecosystem50

EWS-FLI1 and HOXD13 control tumor cell plasticity in Ewing sarcoma

资源简介：

相关数据集