Differential Response Following Spinal Cord Injury in EphA4 Knockout

ChEBI2012-05-23 更新2026-07-21 收录

器官损伤修复

基因敲除模型

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-34430 数据链接链接失效反馈

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Differential Response Following Spinal Cord Injury in EphA4 Knockout

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2012-05-23

The E3 ubiquitin ligase MARCH1 regulates glucose-tolerance and lipid storage in a sex-specific manner

代谢疾病机制

基因敲除模型

Type 2 diabetes is typified by insulin-resistance in adipose tissue, skeletal muscle, and liver, leading to chronic hyperglycemia. Additionally, obesity and type 2 diabetes are characterized by chroni

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Gene expression data from the skin of Cdsniep-/-adult mice. Mus musculus

疾病分子机制

基因敲除模型

Peeling Skin Disease (PSD; OMIM 270300) is an inflammatory ichthyosis due to homozygous loss-of-function mutations in Corneodesmosin (CDSN) and characterized by lifelong patchy peeling of the skin ass

NIAID Data Ecosystem60

Assessing the impact of boldine on the gastrocnemius using multiomics profiling at 7 and 28 days post-complete spinal cord injury in young male mice

器官损伤修复

肌肉萎缩多组学分析

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem40

INSIG1 in NASH pathophysiology and in the hepatic wound-healing response

NASH病理机制

器官损伤修复

We investigated the biological function of INSIG1 in acute (CCl4) and chronic (NASH) liver damage. Male whole-body Insig1 wild-type (WT), Heterozygous (HET) and Knock-out (KO) mice were: 1) challenged

NIAID Data Ecosystem60

Gene expression in the embryonic Lrrc10 knockout heart. Mus musculus

发育基因表达

基因敲除模型

LRRC10 is a heart-specific gene required for proper cardiac function. The effects of Lrrc10 deletion on gene expression in the embryonic mouse heart was investigated. Overall design: Pregnant heterozy

NIAID Data Ecosystem50

Differential Response Following Spinal Cord Injury in EphA4 Knockout

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