Hypertrophic cardiomyopathy (HCM) is one of the most frequent inherited heart condition and a well-established risk factor for cardiovascular mortality worldwide. Although hypertrophy is traditionally
RAD (Ras associated with diabetes) is a membrane protein that acts as a calcium channel regulator by interacting with cardiac L-type Ca2+ channels (LTCC). RAD defects can disrupt intracellular calcium
Analysis of the function of CnAb1 in myocardial hypertrophy in mice using a Gain of Function strategy and Trans Aortic Banding as a hypertrophic stimuly. Microarray was performed after QPCR validation
Cardiovascular diseases (CVD) remain the leading cause of morbidity and mortality worldwide. Arteriolar tone regulation plays a critical role in maintaining appropriate organ blood flow and perfusion
