Intestinal Salmonella Typhimurium infection leads to miR-29a induced Caveolin 2 regulation
收藏NIAID Data Ecosystem2026-03-07 收录
官方服务:
资源简介:
This SuperSeries is composed of the SubSeries listed below. Refer to individual Series
应用场景:
创建时间:
2013-07-10
相关数据集
Type 3 innate lymphoid cells direct goblet cell differentiation via the LT-LTβR pathway during Listeria infection. Type 3 innate lymphoid cells direct goblet cell differentiation via the LT-LTβR pathway during Listeria infection
As a specialized subset of intestinal epithelial cells (IECs), goblet cells (GCs) play an important role during the antibacterial response via mucin production. However, the regulatory mechanisms invo
NIAID Data Ecosystem130
Distinct N6-methyladenosine (m6A) Regulation of Innate Immune Responses During Bacterial Infections II. Distinct N6-methyladenosine (m6A) Regulation of Innate Immune Responses During Bacterial Infections II
N6-methyladenosine (m6A) is a dynamic post-transcriptional RNA modification influencing all aspects of mRNA biology. Here, we examined cellular m6A epitranscriptomes during P.aeruginosa infection to i
NIAID Data Ecosystem80
Table 5_Memory-like CD8+ T cells lacking PD-1 adapt to persistent stimulation by reducing TCR signal transduction rather than increasing exhaustion.xlsx
CD8+ T cells respond to persistent stimulation during chronic viral infection by stably expressing co-inhibitory receptors and other exhaustion-related molecules. Here we addressed how memory-like CD8
NIAID Data Ecosystem50
RIP-seq for SCML2 and SCML2 mutants in 293T-REx and K562 cells
Polycomb repressive complex-1 (PRC1) is essential for the epigenetic regulation of gene expression. SCML2 is a mammalian homolog of Drosophila SCM, a Polycomb-group protein that associates with PRC1.
NIAID Data Ecosystem80
Supplementary Material for: Group A Streptococcus Modulates Host Inflammation by Manipulating Polymorphonuclear Leukocyte Cell Death Responses
Polymorphonuclear leukocyte (PMN) cell death strongly influences the resolution of inflammatory episodes, and may exacerbate adverse pathologies in response to infection. We investigated PMN cell deat
NIAID Data Ecosystem50



