Innate immune cells, such as macrophages, respond to pathogen-associated molecular patterns, such as a lipopolysaccharide (LPS), to secrete various inflammatory mediators. Recent studies have suggeste

NIAID Data Ecosystem40

Phosphatidylcholine treatment significantly downregulates IL-17A and IL-22 production in total and DN ILC3s of NEC mice

炎症免疫调节

ILC3s

Experimental Design Core Objective: Flow cytometry was performed to evaluate the effect of phosphatidylcholine intervention on the effector cytokine production of intestinal lamina propria total group

NIAID Data Ecosystem20

Type I IFNs and TNF Cooperatively Reprogram Epigenomic Landscape of Human Macrophages to Promote Inflammatory Activation

细胞表观遗传学

炎症免疫调节

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem60

Supplementary Material for: Interleukin 8 Elicits Rapid Physiological Changes in Neutrophils That Are Altered by Inflammatory Conditions

中性粒细胞生理机制

炎症免疫调节

A sufficient response of neutrophil granulocytes stimulated by interleukin (IL)-8 is vital during systemic inflammation, for example, in sepsis or severe trauma. Moreover, IL-8 is clinically used as b

Figshare2021-04-15 更新30

Predicated microRNAs that could bind to anti-inflammatory cytokines.

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