Comparison of cancer-stroke patients with or without conventional stroke mechanism.

Figshare2025-06-27 更新2026-04-28 收录

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https://figshare.com/articles/dataset/Comparison_of_cancer-stroke_patients_with_or_without_conventional_stroke_mechanism_/29427118 数据链接链接失效反馈

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资源简介：

Comparison of cancer-stroke patients with or without conventional stroke mechanism.

创建时间：

2025-06-27

Network pharmacology-and molecular docking-based investigation of Danggui blood-supplementing decoction in ischaemic stroke

中药网络药理学

缺血性卒中机制

Danggui blood-supplementing decoction (DBsD) is an herbal preparation treating several diseases including stroke. The present study sought to investigate the potential mechanism of DBsD in ischaemic s

Taylor & Francis Group2024-02-23 更新40

data.pzfx

神经保护药物研发

缺血性卒中机制

Neuroprotective effect of ZLXN in different doses based on neurobehavioral scores and ischemic infarct area. The Longa’s neurological scale was applied to evaluate neurobehavioral deficit in ischemia/

NIAID Data Ecosystem40

Transcriptomic and proteomic multi-model global gene expression analysis reveals regulatory signatures during ischemia stroke (photothrombotic internal ischemia stroke). Transcriptomic and proteomic multi-model global gene expression analysis reveals regulatory signatures during ischemia stroke (photothrombotic internal ischemia stroke)

缺血性卒中机制

基因表达调控

The transcriptomes of focal region in C57BL/6 mice after photothrombotic internal ischemia stroke generated by RNA-sequencing, using Illumina Overall design: mRNA profiles of the micro-punched focal/s

NIAID Data Ecosystem10

Genetic lineage tracing and single cell RNAseq analysis identifies unique feature and cell interactions in resident microglia and infiltrating monocyte derived macrophage at different stages of ischemic stroke

神经炎症细胞异质性

缺血性卒中机制

Ischemic stroke elicits a strong neuroinflammatory response, characterized by the activation of microglia and infiltrating immune cells from the very acute stages onward. Although microglia participat

NIAID Data Ecosystem40

CircFKBP3 absence alleviates oxygen glucose deprivation-induced function loss of human brain microvascular endothelial cells in vitro via governing the miR-766-3p/TRAF3 axis

缺血性卒中机制

circRNA调控

Brain microvascular endothelial cell (BMEC) functions loss is a key event in the development of ischemic stroke, which may be affected by the dysregulation of circular RNAs (circRNAs). We aimed to unv

NIAID Data Ecosystem10

Comparison of cancer-stroke patients with or without conventional stroke mechanism.

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