首页/数据集/The oncogenic BRD4-NUT chromatin regulator drives aberrant transcription within large topological domains

The oncogenic BRD4-NUT chromatin regulator drives aberrant transcription within large topological domains

ChEBI2015-07-14 更新2026-07-12 收录

癌细胞调控

癌基因转录机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-70868 数据链接链接失效反馈

官方服务：

资源简介：

The oncogenic BRD4-NUT chromatin regulator drives aberrant transcription within large topological domains

应用场景：

创建时间：

2015-07-14

相关数据集

TF ChIP-seq from K562 (ENCSR350GZC)

转录因子结合位点

癌细胞调控

eGFP-ZNF561 ChIP-seq on human K562 For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Pages/Research/ENCODE/ENCODE_Data_Use_Policy_for_Exter

NIAID Data Ecosystem60

Gene expression profile analysis of the molecular mechanism of HOXD10 regulating epithelial ovarian cancer cells

癌细胞调控

卵巢癌代谢机制

Epithelial ovarian cancer (EOC) is the most common type of ovarian cancer. Although studies have reported that downregulation of HOXD10 expression may contribute to the migration and invasion abilitie

NIAID Data Ecosystem50

CD95/Fas increases stemness in cancer cells by inducing a STAT1 dependent Type I interferon response [array]

癌细胞调控

干扰素信号通路

CD95 was found to be important for both the formation of CSCs and their maintenance. Using a stable isotope labeling by amino acids in cell culture (SILAC) analysis we have now identified STAT1 as a

NIAID Data Ecosystem40

Transcriptome Analysis of RUVBL1

癌细胞调控

RUVBL1转录组分析

RUVBL1 was shown to be essential for tumor cell growth of many cancers.We identified a lot of genes that are changed obviously by RUVBL1. Gene ontology and Kyoto Encyclopedia of Genes and Genomes anal

NIAID Data Ecosystem60

The Myc binding partner Spt5 is essential for brain tumor growth in Drosophila. The Myc binding partner Spt5 is essential for brain tumor growth in Drosophila

癌细胞调控

果蝇肿瘤模型

The transcription factor SPT5 physically interacts with MYC oncoproteins and is essential for the full transcriptional activation of MYC targets in cultured cells. Here, we use Drosophila to show that

NIAID Data Ecosystem50