首页/数据集/Persistent epigenome anomalies in induced pluripotent stem cells from late-onset sporadic Alzheimer’s disease cases

Persistent epigenome anomalies in induced pluripotent stem cells from late-onset sporadic Alzheimer’s disease cases

NIAID Data Ecosystem2026-05-02 收录

阿尔茨海默病模型

表观遗传学异常

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE255985 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2024-07-30

阿尔茨海默病模型

小胶质细胞表型

Statistical table (See additional statistical table file). Statistical analysis information is displayed for all data displayed within the accompanying figures. Power values were determined post-hoc b

Figshare2016-12-15 更新60

N1-methyladenosine modification in mitochondrial tRNAs is associated with Alzheimer’s disease. N1-methyladenosine modification in mitochondrial tRNAs is associated with Alzheimer’s disease

m1A修饰图谱

阿尔茨海默病模型

Here we used the newly developed m1A-quant-seq appraoch to map m1A modification transcriptome wide on the cortex of 6 month old BL6 mice and 5XFAD mice (Alzheimer's mouse model). Overall design: RNA w

NIAID Data Ecosystem50

Base editor mediated correction of a tau mutation rescues cognitive decline in a mouse model of Alzheimers disease. Homo Sapiens

基因编辑治疗

阿尔茨海默病模型

Low sequence reads

NIAID Data Ecosystem40

Supplementary Material for: Disbalance of the duodenal epithelial cell turnover and apoptosis accompanies insensitivity of intestinal redox homeostasis to inhibition of the brain glucose-dependent insulinotropic polypeptide receptors in a rat model of sporadic Alzheimer's disease

上皮细胞动力学

阿尔茨海默病模型

Medicine

NIAID Data Ecosystem50

Validity and value of metabolic connectivity in mouse models of β-amyloid and tauopathy

阿尔茨海默病模型

代谢关联分析

The goal of the current work was to analyze and validate metabolic regional network alterations in three different AD mouse models (β-amyloid and tau) by use of 2-deoxy-2-[18F]fluoro-d-glucose positro

NIAID Data Ecosystem40

Persistent epigenome anomalies in induced pluripotent stem cells from late-onset sporadic Alzheimer’s disease cases

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