p27Kip1 regulates transcription of genes involved in tumorigenesis

NIAID Data Ecosystem2026-03-11 收录

肿瘤基因调控

细胞周期蛋白

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE20069 数据链接链接失效反馈

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This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

创建时间：

2019-03-04

The structure of the carboxy-terminal domain of DNTTIP1

蛋白质结构解析

细胞周期蛋白

The structure of the carboxy-terminal domain of DNTTIP1 Descriptor: Deoxynucleotidyltransferase terminal-interacting protein 1 Authors: Schwabe, J.W.R, Muskett, F.W, Itoh, T. Deposit date: 2014-11-11

Protein Data Bank Japan2024-05-01 更新70

Reconstructing gene regulatory networks of tumorigenesis [genex]. Homo sapiens

肿瘤基因调控

多组学整合分析

The mechanistic links between transcription factors and the epigenetic landscape, which coordinate the deregulation of gene networks during cell transformation are largely unknown. We used an isogenic

NIAID Data Ecosystem30

Table_1_Downregulation of ZNF280A inhibits proliferation and tumorigenicity of colorectal cancer cells by promoting the ubiquitination and degradation of RPS14.docx

结直肠癌分子机制

肿瘤基因调控

Colorectal cancer (CRC), one of the cancers with highest mortality, involves complicated molecular mechanisms leading to the onset of malignant phenotypes. ZNF280A, a member of the zinc-finger protein

NIAID Data Ecosystem30

The HES1 effects on TCL cells

肿瘤基因调控

淋巴结病理

This study investigated the effects of HES1 depletion on TCL cells with RNA expression profile of two ALK+ TCL cells by Highthrough sequencing RNAseq on SUDHL-1 and SUP-M2 cells with wildtype and HES1

NIAID Data Ecosystem60

The Protein Level of Rev1, a TLS Polymerase in Fission Yeast, Is Strictly Regulated during the Cell Cycle and after DNA Damage

细胞周期蛋白

DNA损伤应答

Translesion DNA synthesis provides an alternative DNA replication mechanism when template DNA is damaged. In fission yeast, Eso1 (polη), Kpa1/DinB (polκ), Rev1, and Polζ (a complex of Rev3 and Rev7) h

NIAID Data Ecosystem30

p27Kip1 regulates transcription of genes involved in tumorigenesis

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