Colony stimulating factor (CSF1) treatment of kidneys that have undergone ischemia reperfusion injury

ChEBI2011-06-20 更新2026-07-21 收录

缺血再灌注损伤

CSF1治疗研究

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Colony stimulating factor (CSF1) treatment of kidneys that have undergone ischemia reperfusion injury

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2011-06-20

Protective role of silent information regulator 1 against hepatic ischemia: effects on oxidative stress injury, inflammatory response, and MAPKs

缺血再灌注损伤

氧化应激与炎症

Objective : Previous studies have verified that silent information regulator 1 (SIRT1), a class III histone deacetylase, protects against ischemia reperfusion (IR) injury (IRI) in some organs. I

DataCite Commons2024-03-24 更新120

Transcriptome Analysis of Renal Ischemia/Reperfusion Injury and its Modulation by Ischemic Pre-Conditioning or Hemin treatment

缺血再灌注损伤

缺血预处理与血红素治疗

We examined, through an oligonucleotide microarray protocol, the renal differential transcriptome profiles of mices submitted to IRI, IPC and Hemin treatment. After identifying the profiles of differe

NIAID Data Ecosystem50

Downregulation of autophagy is associated with severe ischemia-reperfusion-induced acute kidney injury in overexpressing C-reactive protein mice

缺血再灌注损伤

自噬调节机制

C-reactive protein (CRP), was recently reported to be closely associated with poor renal function in patients with acute kidney injury (AKI), but whether CRP is pathogenic or a mere biomarker in AKI r

NIAID Data Ecosystem40

Text S1 - NHE Inhibition Does Not Improve Na + or Ca 2+ Overload During Reperfusion: Using Modeling to Illuminate the Mechanisms Underlying a Therapeutic Failure

缺血再灌注损伤

心血管计算模型

Contains names and constant values (where applicable) for all model variables, all model equations, additional information regarding experiments used to inform model parameters, a summary of the ische

NIAID Data Ecosystem50

Data_Sheet_1_Human Recombinant Alkaline Phosphatase (Ilofotase Alfa) Protects Against Kidney Ischemia-Reperfusion Injury in Mice and Rats Through Adenosine Receptors.PDF

缺血再灌注损伤

药物干预机制

Adenosine triphosphate (ATP) released from injured or dying cells is a potent pro-inflammatory “danger” signal. Alkaline phosphatase (AP), an endogenous enzyme that de-phosphorylates extracellular ATP

NIAID Data Ecosystem40

Colony stimulating factor (CSF1) treatment of kidneys that have undergone ischemia reperfusion injury

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