Metabolomic and Transcriptomic Signatures of Chemnogenetic Heart Failure

Alliance of Genome Resources2026-08-01 收录

心力衰竭转录组学

代谢组学心脏疾病

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https://www.ncbi.nlm.nih.gov/gds/?term=GSE195913 数据链接链接失效反馈

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In our chemogenetic rat heart failure model, LV and RV transcriptomic analysis was performed by RNA-seq

Chamber-Enriched gene expression profiles in failing human hearts with reduced ejection fraction [RNA-Seq]. Chamber-Enriched gene expression profiles in failing human hearts with reduced ejection fraction [RNA-Seq]

心力衰竭转录组学

心脏疾病分子机制

Heart failure with reduced ejection fraction (HFrEF) constitutes 50% of HF hospitalizations and is characterized by high rates of mortality. To explore the underlying mechanisms of HFrEF etiology and

NIAID Data Ecosystem40

Transcriptional profiling of human white blood cells at the different stages of NYHA classification. Homo sapiens

心力衰竭转录组学

心衰阶段分子标志物

Human white blood cells from healthy, heart failure risk patients, asymptomatic left ventricular dysfunction patients, chronic heart failure, acute heart failure patients Overall design: Common refere

NIAID Data Ecosystem30

ReHeaT

心力衰竭转录组学

生物标志物发现

ReHeaT is a user friendly portal to explore, analyze and download the transcriptional consensus signature of human end-stage heart failure (HF-CS). The HF-CS is based on a meta-analysis of 16 independ

知名数据库10

Early activated ageing-related changes in cardiac transcriptome of Tgaq*44 mice during entire HF development

心脏老化分子机制

心力衰竭转录组学

To investigate ageing-related changes in cardiac transcriptome of FVB mice we performed differential gene expression profiling analysis using data obtained from RNA-seq of five life time points (4-, 8

NIAID Data Ecosystem40

changes in cardiac transcription profiles brought about by heart failure

心力衰竭转录组学

抗病分子机制

To establish changes in cardiac transcription profiles brought about by heart failure we collected myocardial samples from patients undergoing cardiac transplantation whose failure arises from differe

NIAID Data Ecosystem20

Metabolomic and Transcriptomic Signatures of Chemnogenetic Heart Failure

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