Expression profiling of mammalian Schwann cells in response to treatment with NRG and/or IGF

Alliance of Genome Resources2026-08-01 收录

线粒体生物发生

IGF-1信号通路

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https://www.ncbi.nlm.nih.gov/gds/?term=GSE10775 数据链接链接失效反馈

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Keywords: Input of growth and mitogenic pathways in mitochondria biogenesis

Insulin-like growth factor 1 (IGF1) heart study

心肌分子机制

IGF-1信号通路

IGF1 and IGF1 receptors (IGF1R) are present in the adult heart and have been shown to be essential for myocardial performance. Insulin-like growth factor 1 (IGF1) is produced in numerous tissues parti

NIAID Data Ecosystem30

IGF-1, IGF-1receptor mRNA, and L-[U-14C]-phenylalanine incorporation into proteins, in confluent non-proliferating L6 skeletal muscle cells.

骨骼肌代谢

IGF-1信号通路

Cells were amino acid refed at High AA concentrations (9 mM) with or without presence of inhibitors of molecules in the IGF-1receptor signaling pathway (mean±SE, n = 4-5/group).

NIAID Data Ecosystem20

Artemisinin mimics calorie restriction to trigger mitochondrial biogenesis and compromise telomere shortening in mice

线粒体生物发生

端粒动力学

Calorie restriction is known to extend lifespan among organisms by a debating mechanism underlying nitric oxide-driven mitochondrial biogenesis. We report here that nitric oxide generators including a

NIAID Data Ecosystem20

Yeast Mitochondrial Biogenesis: A Role for the PUF RNA-Binding Protein Puf3p in mRNA Localization

线粒体mRNA定位调控

线粒体生物发生

The asymmetric localization of mRNA plays an important role in coordinating posttranscriptional events in eukaryotic cells. We investigated the peripheral mitochondrial localization of nuclear-encoded

NIAID Data Ecosystem20

FOXO and HSF1 rejuvenate immunity via positive feedback regulation of insulin/IGF-1 signaling

免疫衰老

IGF-1信号通路

A hallmark of aging is immunosenescence, a decline in immune function that appeared to be inevitable. Surprisingly, here we show that genetic inhibition of DAF-2/insulin/IGF-1 receptor drastically del

NIAID Data Ecosystem20

Expression profiling of mammalian Schwann cells in response to treatment with NRG and/or IGF

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