首页/数据集/H3K4 trimethylation ChIP - ctk1 deletion

H3K4 trimethylation ChIP - ctk1 deletion

NIAID Data Ecosystem2026-03-07 收录

表观遗传调控

组蛋白修饰定位

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE4533 数据链接链接失效反馈

官方服务：

资源简介：

Genome-wide location analysis of histone H3 lysine-4 trimethylation in a ctk1 deletion strain Keywords: repeat Genome-wide location analysis of histone H3 lysine-4 trimethylation in a ctk1 deletion strain

应用场景：

创建时间：

2012-03-17

相关数据集

H3K9acS10P (new abcam lot).Kc

组蛋白修饰定位

ChIP-seq分析

modENCODE_submission_3009 This submission comes from a modENCODE project of Gary Karpen. For full list of modENCODE projects, see http://www.genome.gov/26524648 Project Goal: We aim to determine the l

NIAID Data Ecosystem30

Addressing the specific roles of histone modifications in transcriptional repression [RNA-seq]

组蛋白修饰转录抑制

表观遗传调控

Our understanding of epigenetic processes is based on the hypothesis that individual posttranslational modifications of DNA and histones, or combinations thereof, function to direct unique downstream

NIAID Data Ecosystem00

Mitochondrial Fission 1 (FIS1) knockdown effect on the H3K27Ac status of leukemia cell line MOLM-13 cells. Mitochondrial Fission 1 (FIS1) knockdown effect on the H3K27Ac status of leukemia cell line MOLM-13 cells

表观遗传调控

肾病与白血病基因功能研究

To examine if loss of FIS1 would alter the H3K27Ac status of leukemic cells, we used shRNA to knock down the expression of FIS1 in leukemic cell line MOLM-13 cells and performed H3K27Ac CHIP-seq to ch

NIAID Data Ecosystem00

DNMT3A Reads and Connects Histone H3K36me2 to DNA methylation [RNA-seq]

表观遗传调控

甲基化反应机制

DNA methylation at the 5-position of cytosine (5mC) is a crucial epigenetic mark in regulating biological processes including gene silencing, gene imprinting, and X chromosome inactivation. It has lon

NIAID Data Ecosystem00

An epigenetic switch in vascular phenotype augments anti-tumor immunity [bulkRNA-seq]

肿瘤免疫微环境

表观遗传调控

The abnormal tumor vasculature can present a barrier to the infiltration of anti-tumor immune cells, which impairs immune surveillance and response to immunotherapy. Here, we show that targeting the e

NIAID Data Ecosystem00