MSH4/MSH5 are critical components of the class I crossover (CO) machinery, which is responsible for >90% of the COs that arise in mammalian meiosis. We generated a point mutation in the ATP binding mo
These data map crossover recombination events in N2/CB4568 heterozygous animals. We show that crossover recombination events are doubled in syp-4(ie25) mutants compared to wild-type animals.
