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Trichuris muris infection drives cell-intrinsic IL4R alpha independent large intestinal RELMα+ macrophages.

ChEBI2021-11-15 更新2026-07-12 收录

寄生虫免疫

巨噬细胞表型分析

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-10501 数据链接链接失效反馈

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Trichuris muris infection drives cell-intrinsic IL4R alpha independent large intestinal RELMα+ macrophages.

应用场景：

创建时间：

2021-11-15

寄生虫免疫

宿主细胞转录组

A protein of Eimeria tenella (encoded by the locus ETH_00028350) homologous to Toxoplasma gondii dense granule protein 9, designated as EtHGRA9 hereafter, was reported to be expressed in all life cycl

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Influenza virus-like particles presenting Toxoplasma gondii dense granule protein 7 protect mice from lethal ME49 challenge

弓形虫疫苗

寄生虫免疫

Toxoplasma gondii dense granule antigen 7 (GRA7) is a membrane-associated protein expressed across parasite life cycle and represents a promising vaccine target. This study aimed to develop a GRA7-bas

Figshare2025-08-13 更新50

Additional file 6 of NR4A1 deletion promotes pro-angiogenic polarization of macrophages derived from classical monocytes in a mouse model of neovascular age-related macular degeneration

巨噬细胞表型分析

年龄相关性黄斑变性

Additional file 6: Table S4. Differential ADT expression for each cluster from mononuclear phagocytes. Avg_log2FC = log2 fold change, pct.1 = percent of cells in cluster that express the gene, pc.2 =

Figshare2024-08-14 更新40

Immune evasion and the evolution of molecular mimicry in parasites

寄生虫免疫

分子进化

Parasites that are molecular mimics express proteins which resemble host proteins. This resemblance facilitates immune evasion because the immune molecules with the specificity to react with the paras

NIAID Data Ecosystem60

Counts of CD68+ cells, CD68+CD80+ cells, CD68+CD86+ cells and CD68+PD-L1+ cells.

器官免疫微环境

巨噬细胞表型分析

The average count ± S.D. for each areas of the liver is represented as indicated. The p value indicates the significance of differences between HBV-infected and control groups.

Figshare2016-09-28 更新40

Trichuris muris infection drives cell-intrinsic IL4R alpha independent large intestinal RELMα+ macrophages.

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