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Supplementary Table S3 from Tumor Microenvironment Remodeling Enables Bypass of Oncogenic KRAS Dependency in Pancreatic Cancer

NIAID Data Ecosystem2026-04-25 收录

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基因编辑技术应用

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https://figshare.com/articles/dataset/Supplementary_Table_S3_from_Tumor_Microenvironment_Remodeling_Enables_Bypass_of_Oncogenic_KRAS_Dependency_in_Pancreatic_Cancer/22535674 数据链接链接失效反馈

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shRNA and sgRNA information

应用场景：

创建时间：

2020-07-01

癌症分子机制

依赖性基因调控

Additional file 3. Overexpression of ALKBH5 decreased the proliferation capacity in 2 HCC cell lines, while co-overexpressed of PAQR4 reverted this phenomenon (A and B). Overexpression of ALKBH5 decre

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β-arrestin1 is involved in hepatocellular carcinogenesis via an inflammation-mediated Akt signal

癌症分子机制

基因表达谱分析

Hepatocellular carcinoma (HCC), the main form of liver cancer, is the sixth most common cancer and the third most frequent cause of cancer death worldwide1. The exact mechanism of HCC initiation and d

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A renal cancer-associated, renal tubule-specific, HIF-binding enhancer of oncogenic MYC and PVT1 expression. Homo sapiens

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MYC转录因子

Clear cell renal cell carcinoma is characterized by loss of function of the von Hippel-Lindau tumor suppressor gene (VHL) and unrestrained activation of hypoxia inducible transcription factors (HIF).

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WTX overexpression effect on gastric cell line

癌症分子机制

肿瘤抑制基因

We used microarrays to detail the global gene expression in AGS gastric cancer cells overexpressing WTX, a tumor suppressor frequently inactivated in Wilms tumor. Results provide insight into roles fo

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DataSheet4_PRR11 promotes cell proliferation by regulating PTTG1 through interacting with E2F1 transcription factor in pan-cancer.zip

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细胞周期调控

The upregulated proline rich 11 (PRR11) plays a critical role in cancer progression. The relevant biological functions of PRR11 in pan-cancer development are not well understood. In the current study,

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Supplementary Table S3 from Tumor Microenvironment Remodeling Enables Bypass of Oncogenic KRAS Dependency in Pancreatic Cancer

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